学术会议海报模板

横向学术poster模板
横向学术海报（poster）是一种常见的学术交流形式，用于展示研究成果、观点或实验数据。

以下是一个横向学术海报的模板，包含了一般情况下需要包括的几个主要部分：
1. 标题部分：
海报标题，简洁明了地概括研究内容或主题。

作者信息，包括作者姓名、所属机构和联系方式。

2. 引言部分：
研究背景，介绍研究领域的背景和相关文献。

研究问题，明确研究的目标和问题。

研究意义，阐述研究的重要性和应用价值。

3. 方法部分：
研究设计，描述实验设计、数据收集和分析方法。

样本/实验对象，介绍研究所使用的样本或实验对象。

测量指标，列出用于评估研究结果的主要指标。

4. 结果部分：
数据展示，通过图表、表格或图片等方式展示研究结果。

结果解读，对结果进行解读和分析，突出关键发现和趋势。

5. 讨论部分：
结果解释，解释结果与研究问题之间的关系。

结果比较，将结果与相关研究进行比较和讨论。

结果影响，讨论结果对该领域的影响和可能的应用。

6. 结论部分：
主要结论，总结研究的主要发现和结论。

展望未来，提出进一步研究的方向和建议。

7. 参考文献部分：
引用文献，列出在海报中引用的相关文献。

此外，还可以根据具体研究内容的需要添加其他部分，如实验
过程、图示说明等。

请注意，以上仅是一个一般的横向学术海报模板，具体的内容
和结构可以根据研究的具体要求和学术会议的规定进行调整和修改。

Results Of the 96 investigated genes, 9 were significantly up-regulated in the high inflammation group of samples. Four of them were significantly correlated with the inflammation score: CCR7, CD40LG, CTLA4 and ICOS (the IL17 up-regulation was not significant). In the urine samples, ICOS protein expression was easily quantified by ELISA and was associated with a higher post-void residual (93.2 vs 37.2ml; p=0.020) and a lower maximum urinary flow rate (9.1 vs 12.7ml/s; p=0.033). Grégoire ROBERT 1,2, Frank SMIT 3, Kees JANSEN 1, Tilly AALDERS 1, Alexandre de la TAILLE 4, Jack SCHALKEN 11. Radboud University Nijmegen Medical Center, Experimental Urology, Nijmegen, The Netherlands2. Bordeaux Pellegrin University Hospital, Department of Urology, Bordeaux, France3. Noviogendix, Nijmegen, The Netherlands4. Henri-Mondor Hospital, AP-HP , Department of Urology, Créteil, FranceIntroduction and ObjectivesChronic prostatic inflammation (CPI) may be a central mechanism in prostateenlargement and benign prostatic hyperplasia (BPH) progression 1, 2.Until today CPI can only be diagnosed by prostatic biopsies. But to furtherinvestigate the link between CPI and BPH in clinical trials, a less invasivediagnostic tool in needed.As a first step towards the identification of non invasive biomarkers forprostatic inflammation, our objectives were:study the inflammatory and immune responses-To correlate the mRNA expression with an histological inflammation score-To investigate the protein expression of these potential biomarkers in urinesamplesMaterial and Methods90 BPH tissue samples were investigated in two steps.First a hypothesis was generated using a profiling procedure with a panel of96 genes on an initial set of 30 BPH samples.Then the candidate biomarkers were validated on a large number of samples(n=90).A histological inflammation score based on the density and the confluence oflymphoid inflammation.The relative expression of the genes were calculated and compared with theFigure 1: Quantitative real-time PCR gene target relative quantification (RQ) in 90 BPH samples according to the histological inflammation score (none (n=6), mild (n=19), moderate (n=39) or severe (n=26) prostatic inflammation) Figure 2: Post-void residual and maximum uroflowmetry according to the ICOS protein expression (<357 pg/ml versus >357 pg/ml) in 45 urine samples collected after digital rectal examination Conclusions Four genes were significantly up regulated at the mRNA level in the prostatic tissue of patients with severe inflammation: CCR7, CD40LG, CTLA4 and ICOS. In urine, and at the protein level, ICOS was easily measured by ELISA and was associated with a higher post-void residual and a lower maximum urinary flow rate.G. Robert has been granted by the EAU Scholarship Program (EUSP grant S03-2009) and by the French Urological Association (Association Française d’Urologie - AFU) References: 1. Nickel JC et al: The Relationship between Prostate Inflammation and Lower Urinary Tract Symptoms: Examination of Baseline Data from the REDUCE Trial. Eur Urol 2008 2. Robert G et al. Inflammation in benign prostatic hyperplasia: a 282 patients' immunohistochemical analysis. The Prostate 2009。